Clenbuterol Rapid Test (0.5ppb)

Clenbuterol hydrochloride is a hydrochloride that is the monohydrochloride salt of clenbuterol. It has a role as a bronchodilator agent, a beta-adrenergic agonist and a sympathomimetic agent.
A substituted phenylaminoethanol that has beta-2 adrenomimetic properties at very low doses. It is used as a bronchodilator in asthma.

Soluble in water and in ethanol (96 per cent), slightly soluble in acetone.

Clenbuterol hydrochloride is chemically named α-[(tert-butylamino) methyl] -4-amino-3,5-dichlorobenzyl alcohol hydrochloride, with a molecular formula of C12H18CI2N2O · HCI and a molecular weight of 313.65. It is a white or similar white crystalline powder, odorless, bitter, soluble in water, ethanol, slightly soluble in acetone or chloroform, insoluble in ether, chemically stable, cannot be destroyed by ordinary methods, and heated to 172 ° C to break down. Clenbuterol is one of the synthetic β-adrenergic receptor stimulants. It is a powerful agonist. It has a strong and long-lasting effect on bronchial smooth muscle. It is mainly used in the treatment of asthma and has asthma effect. Generally, 30pg can work on the human body.

It binds to the β-adrenergic receptor to form an agonist receptor complex, which activates the G5 protein, and the alpha subunit of the G5 protein subsequently activates adenylate cyclase, the cyclic adenylate (cAMP) produced by the enzyme) Is an important intracellular messenger molecule. The mechanism by which cyclic adenosine (cAMP) produces an effect is through binding to the regulatory subunit of protein kinase k to release the catalytic subunit of the enzyme, which phosphorylates a series of intracellular proteins. Some of these proteins are enzymes that are activated when phosphorylated. Cyclic adenylate response element binding protein (CREB) is phosphorylated by protein kinase A, binds to the cAMP response element of a gene regulatory site, and activates the transcription of this gene. Phosphorylation enhances the transcriptional activity of CREB, which is also breastfeeding in animal cells, β-adrenergic receptor agonists mediate the transcription of many genes, and the activities of other enzymes are inhibited during phosphorylation.

Clenbuterol hydrochloride tablets is a synthetic β2-stimulant, which has the function of bronchiectasis and is commonly used to prevent and treat lung diseases such as asthma and emphysema. Large doses can increase myocardial contractility, increase heart rate, and increase myocardial oxygen consumption. It can cause myocardial ischemic injury and even myocardial infarction in patients with heart disease, especially coronary heart disease.

Clenbuterol should be started at a dose of 20mcg. Higher doses can be used after tolerance is assessed but caution is warranted at higher doses due to the potency of the drug. Under no circumstance should one consume more than 120mcg a day.

Clenbuterol hydrochloride can act on a variety of target organs, and excessive and prolonged use will cause chronic toxicity to the organs. The mice were exposed to poison for 30 days, and their right ventricles were dissected and the heart shape was oval. The apex is obtuse, the heart wall becomes thin and soft, and a large amount of blood accumulates in the heart. The heart wall collapses after the heart is opened, and the shape of the heart tissue changes significantly.

Clenbuterol hydrochloride toxicity to the kidney is mainly manifested in high doses that can change the coefficient of the renal organs. At the same time, experiments have shown that prolonged adrenocorticoid proliferation in rats, suggesting that long-term administration of clenbuterol can lead to excessive adrenal secretion and adversely affect the body.

Acute poisoning of clenbuterol hydrochloride is mainly due to the activation of β2 adrenergic receptors in the heart and skeletal muscle. If you take too much clenbuterol for a short period of time, it can cause acute poisoning. The clinical manifestations are heart palpitations, chest tightness, dizziness, headache, nausea and vomiting, sore neck and back muscles, and increased blood pressure. The electrocardiogram showed sinus tachycardia, paroxysmal supraventricular tachycardia, sinoatrial block, and premature beats. Laboratory tests include increased blood image, myocardial enzymes, elevated blood glucose, decreased blood potassium, increased creatinine, and increased urea nitrogen.

In the early 1960s, some people discovered that it could promote animal growth. In the 1970s, the United States introduced it to livestock and poultry production and became the most widely used β-stimulant in the world. However, due to its strong thermal and biochemical stability, animals can form accumulation residues in the internal organs and tissues after eating. Humans who eat lean pork containing chemicalbook fines will have symptoms of poisoning. Therefore, countries around the world have banned the use of β-stimulants in animal husbandry in the near future. Many countries have developed detection methods for monitoring β-stimulant residues and clenbuterol hydrochloride in feed. Chinese government explicitly banned clenbuterol as a feed additive in 1997.

In order to ensure the quality and safety of livestock products and protect human health, many countries have banned the use of clenbuterol in the production of food-borne animals. The US Food and Drug Administration (FDA) has classified clenbuterol in meat Residue is a mandatory inspection item, and the EU is strictly forbidden to add "lean extract" drugs to feed. Although China proposed to ban the use of "clenbuterol" drugs in 2000, the use of "clenbuterol" in animal husbandry production is still banned.
The Clenbuterol Rapid Test (0.5ppb) kit provided by BALLYA can effectively detect whether food contains clenbuterol residues. This kit is not only simple to operate, but also has a short test time and high accuracy. Can ensure consumers food safety.

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